图没看懂？

Effect on Po2/Ventilation Response Curve

The normal relationship between Po2 and ventilation was described on pages 50 et seq. It was long believed that this reflex was the ultimum moriens and, unlike the Pco2/ventilation response curve, unaffected by anaesthesia. This doctrine was a source of comfort to many generations of anaesthetists until the 1970s, when halothane anaesthesia was shown to reduce the acute hypoxic ventilatory response (AHVR) in humans. It was soon shown that not only was the hypoxic response affected by inhalational anaesthetics it was also, in fact, exquisitely sensitive (Fig. 21.3).5 Hypoxic drive was markedly attenuated at 0.1 MAC, a level of anaesthesia that would not be reached for a considerable time during recovery from anaesthesia. Similar effects were found with all the currently used inhalational agents and propofol.

对PO₂/通气反应曲线的影响^3,4

PO₂/通气反应曲线在第50页及之后部分中描述。长期以来，人们一直认为PO₂/通气反应曲线与PCO₂/通气反应曲线不同，缺氧兴奋通气是原始反射，PO₂/通气反应曲线不受麻醉的影响（译者注：moriens，拉丁单词，就是致死性。ultimum moriens为last to be dead，最后死者，It was long believed that this reflex was the ultimum moriens and, unlike the Pco2/ventilation response curve, unaffected by anaesthesia.原文说缺氧兴奋通气是ultimum moriens，不受麻醉的影响，因为原始反射也是不受别的影响的，是死时才消失，考虑到这里讲的也是一种反射，所以将其翻译成“原始反射”）。在20世纪70年代前，这一学说一直是麻醉医师的实践依据，直到70年代证明了氟烷麻醉可降低人类的急性低氧通气反应(acute hypoxic ventilatory response，AHVR)。研究很快表明，吸入麻醉不仅影响低氧反应，而且低氧反应实际上对吸入麻醉也极为敏感（图.21.3）⁵，因为缺氧兴奋通气在0.1MAC深度的吸入麻醉时就明显减弱，而即使在麻醉恢复时相当长的时间内麻醉深度都会高于0.1MAC。目前使用的所有吸入麻醉剂和丙泊酚中均发现了相似的对PO₂/通气反应曲线的抑制³。

Percentage of control ventilation控制通气百分比、End-tidal PO₂呼气末PO_2、Control conditions控制情况、Awake清醒、halothane氟烷

上面文字好理解，就是图片不好理解，什么叫“Percentage of control ventilation控制通气百分比”，怎么会有200%，“Awake清醒”反而“控制通气百分比”高，麻醉时反而“控制通气百分比”低，还有那个状态的变化时（就是那个清醒时曲线的突然下降）啥意思

“Control conditions控制情况”翻译的对吗？