上面的都表示还原态了？

When the mitochondrial Po2 is reduced, oxidative phosphorylation continues normally down to a level of about 0.3 kPa (2 mmHg). Below this level, oxygen consumption falls, and the various members of the electron transport chain tend to revert to the reduced state. Reduced to oxidised nicotinamide adenine dinucleotide (NADH/NAD+) and lactate/ pyruvate ratios rise, and the ATP/ADP ratio falls. The critical Po2 varies between different organs and different species, but, as an approximation, a mitochondrial Po2 of about 0.13 kPa (1 mmHg) may be taken as the level below which there is serious impairment of oxidative phosphorylation and a switch to anaerobic metabolism. Tissue hypoxia is discussed further on page 277. The critical Po2 for oxidative phosphorylation is also known as the Pasteur point, and has applications beyond the pathophysiology of hypoxia in man. In particular, it has a powerful bearing on putrefaction, many forms of which are anaerobic metabolism resulting from a fall of Po2 below the Pasteur point in, for example, polluted rivers

线粒体PO2下降，直到0.3 kPa（2 mmHg）水平前，氧化磷酸化都正常。低于0.3 kPa（2 mmHg）水平时，氧耗量下降，电子传递链的各组分都趋于恢复到还原态。还原型烟酰胺腺嘌呤二核苷酸（NADH）被氧化成氧化型烟酰胺腺嘌呤二核苷酸（NAD+），乳酸/丙酮酸比升高，ATP/ADP比下降。不同器官和不同物种的临界氧分压不同，粗略估计线粒体PO2约为0.13 kPa（1 mmHg）时，可视为氧化磷酸化严重受损和向无氧代谢转换的水平。组织缺氧将在277页深入讨论。氧化磷酸化的临界氧分压也称为巴斯德点，其应用超出了人类缺氧的病理生理范畴。特别是，它对腐败（作用）有很大影响，诸多形式的腐败（作用）是由于PO2低于巴斯德点导致无氧代谢，如在污染河流中的腐败（作用）。

前一句说：电子传递链的各个成员倾向于回到还原状态？上面的都表示还原态了？Reduced to oxidised nicotinamide adenine dinucleotide (NADH/NAD1)怎么理解？会不会是：氧化型烟酰胺腺嘌呤二核苷酸（NAD+）还原成还原型烟酰胺腺嘌呤二核苷酸（NADH）？