有点怪？

A 28 - year - old woman with cystic fibrosis has been followed up in the adult outpatient clinic for the past 12 years.Last year she was admitted to the hospital with severe weakness and lethargy after an episode of upset stomach associated with nausea and loss of appetite. There was no vomiting or diarrhea. Her vital signs on admission showed a blood pressure of 90/55 mm Hg and pulse rate of 90 beats/min. Blood chemical analysis showed Na+, 134 mEq/L; K+, 2.4 mEq/L; HCO3−, 35 mEq/L; Cl−, 88 mEq/L; SUN, 38 mg/dL; and Scr, 1.4 mg/dL. She is not on any diuretics. VBG indicated a pH of 7 .48 and PCO2 of 48 mm Hg. Urine electrolyte profile showed Na+, 35 mEq/L; Cl−, 30 mEq/L; and K+, 28 mEq/L.

一名囊性纤维化的28岁女性在成人门诊随访了12年。去年，她因胃部不适伴恶心和食欲不振后重度无力和嗜睡入院。无呕吐、腹泻。入院时生命体征显示血压90/55 mmHg，脉率90次/min。血生化分析显示Na⁺ 134mEq/L、K⁺ 2.4 mEq/L、HCO₃^-35 mEq/L、Cl^- 88 mEq/L、SUN 38 mg/dL、Scr 1.4 mg/dL。她未使用任何利尿剂。VBG显示pH值为7.48，PCO₂为48 mmHg。尿电解质检查显示Na⁺ 35 mEq/L、Cl^- 30 mEq/L、K⁺ 28 mEq/L。

这里用的多数都是过去时，也就是说在分析去年发病事的病情？

The generation of metabolic alkalosis, volume depletion, and renal Cl− loss in this patient could be best explained by: 

a) Severe volume depletion due to the excessive loss of Na+ and Cl− in sweat 

b) Kidney salt wasting due to excessive consumption of electrolyte replacement solutions 

c) Posthypercapnic metabolic alkalosis after the treatment for respiratory acidosis 

d) The inability to conserve Cl− in the kidney during volume depletion

代谢性碱中毒、血容量不足、肾脏Cl^-丢失最可能的解释是：

a）由于汗液中Na⁺和Cl^-的过量丢失导致血容量严重不足

b）由于过量使用电解质置换液导致的肾脏盐耗

c）呼吸性酸中毒治疗后的高碳酸学症后的代谢性碱中毒

d）容量耗竭时肾脏不能保存Cl^-

呼吸性酸中毒治疗后的高碳酸学症后的代谢性碱中毒，怎么感觉这么奇怪呢，是不是就是呼吸性酸中毒后的代偿性代谢性碱中毒